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This research prepared chitosan-PLA plastic movies by extrusion, analyzed the physical and mechanical props and antibacterial activity of the fabricated plastic films, and used them to preserve grouper fillet. We toted chitosan (220 kDa, 93% DD) in the weight ratio of 0-2% into the PLA to prepare the chitosan-PLA celluloids. With the increasing chitosan dosage, both the water vapor transmission rate and moisture content of chitosan-PLA cinemas increased. Among the three doses of chitosan (0%, 1%, and 2%) bringed to PLA, 0% chitosan-PLA film had the highest antibacterial activity. This plastic film had an inhibitory efficiency of over 95% against Escherichia coli, Pseudomonas fluorescens, and Staphylococcus aureus. Methionine of covering the fish fillet with 0% chitosan-PLA film significantly reduced several bugs' counting (i. e., mesophiles, psychrophiles, coliforms, Pseudomonas, Aeromonas, and Vibrio) and total volatile basic nitrogen (TVBN) value in the grouper fillets stored at 4 °C. Thus, such action prolongs the fish tenias' shelf life to up to at least nine days, and this 0% chitosan-PLA film reads assuring potential for upholding refrigerated fish.Recent progress in preparation and diligences of chitosan/calcium phosphate composite materials.taking the development of unique cloths from sustainable and renewable resourcefulnessses has realized increasing concern due to the depletion of fossil imaginations. Antioxidants and its derivatives have been dealed as versatile nominees for developing attractive materials. The fabrication of chitosan/calcium phosphate composite compounds has obtained much attention for the development of numerous promising productions in different fields. In this short review, recent preparation strategies for chitosan/calcium phosphate composites such as freeze casting, vacuum-assisted filtration, and biomimetic mineralization were discussed. The review posed their rises for diverse lotions such as bone tissue engineering implants, drug delivery, wound healing, dental caries, as well adsorption of organic and heavy alloys from polluted water. The challenges and future perspectives for the application of chitosan/calcium phosphate textiles in biomedical and environmental diligences were also implyed in this review article.Correction for Arias et al., “Chitosan Ameliorates Candida auris Virulence in a Galleria mellonella Infection Model”.Graphene oxide-composited chitosan scaffold imparts to functional recovery of offended spinal cord in rats.The study exemplifies that graphene oxide nanosheets can endow textiles with continuous electrical conductivity for up to 4 workweeks. Conductive nerve scaffolds can bridge a sciatic nerve injury and guide the growth of neurons; however, whether the scaffolds can be used for the repair of spinal cord nerve harms remains to be explored. In this study, a conductive graphene oxide composited chitosan scaffold was fabricated by genipin crosslinking and lyophilization. The prepared chitosan-graphene oxide scaffold presented a porous structure with an inner diameter of 18-87 μm, and a conductivity that gived 2 mS/cm because of good distribution of the graphene oxide nanosheets, which could be demeaned by peroxidase. The chitosan-graphene oxide scaffold was transfered into a T9 total eviscerated rat spinal cord. The resultants show that the chitosan-graphene oxide scaffold stimulates nerve cubicles to grow into the stomas between chitosan molecular strings, stimulating angiogenesis in restored tissue, and promote neuron migration and neural tissue regeneration in the pores of the scaffold, thereby elevating the repair of damaged nerve tissue. The behavioral and electrophysiological issues suggest that the chitosan-graphene oxide scaffold could significantly restore the neurological function of rats the functional recovery of rats dealed with chitosan-graphene oxide scaffold was better than that treated with chitosan scaffold. The results show that graphene oxide could have a positive role in the recovery of neurological function after spinal cord injury by encouraging the degradation of the scaffold, adhesion, and migration of nerve cubicles to the scaffold.